Crosstalk between Cdk5/p35 and ERK1/2 signalling mediates spinal astrocyte activity via the PPARγ pathway in a rat model of chronic constriction injury-Reference-Cited by-同舟云学术

Crosstalk between Cdk5/p35 and ERK1/2 signalling mediates spinal astrocyte activity via the PPARγ pathway in a rat model of chronic constriction injury

Published:2019-09-09 Issue:2 Volume:151 Page:166-184
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J. Neurochem.

Author:

Zhong Yu¹,Chen Jialin¹,Chen Jing¹,Chen Yanhua²,Li Li¹,Xie Yubo¹^ORCID

Affiliation:

1. Department of Anesthesiology The First Affiliated Hospital of Guangxi Medical University Nanning Guangxi P. R. China

2. Department of Anesthesiology Cardiovascular Institute Nanning Guangxi P. R. China

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jnc.14827

Reference72 articles.

1. Cyclin-Dependent Kinase 5/p35/p39: A Novel and Imminent Therapeutic Target for Diabetes Mellitus

2. An ERK/Cdk5 axis controls the diabetogenic actions of PPARγ

3. A peripheral mononeuropathy in rat that produces disorders of pain sensation like those seen in man

4. The role of MAPKs in adipocyte differentiation and obesity

5. Spinal IFN-γ-induced protein-10 (CXCL10) mediates metastatic breast cancer-induced bone pain by activation of microglia in rat models

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