Changes in intrinsic inhibition in isolated hippocampal slices during ethanol withdrawal; lack of correlation with withdrawal hyperexcitability-Reference-Cited by-同舟云学术

Changes in intrinsic inhibition in isolated hippocampal slices during ethanol withdrawal; lack of correlation with withdrawal hyperexcitability

Published:1992-10 Issue:2 Volume:107 Page:521-527
ISSN:0007-1188
Container-title:British Journal of Pharmacology
language:en
Short-container-title:

Author:

Whittington M.A.,Little H.J.,Lambert J.D.C.

Publisher

Wiley

Subject

Pharmacology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1476-5381.1992.tb12777.x/fullpdf

Reference38 articles.

1. Ethanol potentiates the GABAA-activated Cl−current in mouse hippocampal and cortical neurones;AGUEYO;Eur. J. Pharmacol.,1990

2. Acute and chronic ethanol treatments alter GABA receptor-operated chloride channels;ALLAN;Pharmacol. Biochem. Behav.,1987

3. Ethanol potentiates GABA- and glycine-induced chloride currents in chick spinal cord neurones;CELENTANO;Brain Res.,1988

4. Antagonism of the enhanced susceptibility to audiogenic seizures during alcohol withdrawal in the rat by gamma-aminobutyric acid (GABA) and ‘GABA-mimetic’ agents;COOPER;J. Pharmacol. Exp. Ther.,1979

5. Paired pulse and frequency facilitation in the CA1 region of the in vitro rat hippocampus;CREAGER;J. Physiol.,1980

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