Acute and chronic ethanol treatments alter GABA receptor-operated chloride channels-Reference-Cited by-同舟云学术

Acute and chronic ethanol treatments alter GABA receptor-operated chloride channels

Published:1987-08 Issue:4 Volume:27 Page:665-670
ISSN:0091-3057
Container-title:Pharmacology Biochemistry and Behavior
language:en
Short-container-title:Pharmacology Biochemistry and Behavior

Author:

Allan Andrea M.,Harris R.Adron

Publisher

Elsevier BV

Subject

Behavioral Neuroscience,Biological Psychiatry,Clinical Biochemistry,Pharmacology,Toxicology,Biochemistry

Reference42 articles.

1. Ethanol and barbiturates enhance GABA-stimulated influx of 36Cl− in isolated brain membranes;Allan;Pharmacologist,1985

2. Gamma-aminobutyric acid and alcohol actions: Neurochemical studies;Allan;Life Sci,1986

3. Ethanol tolerance measured electrophysiologically in hippocampal slices and not in neuromuscular junctions from chronically ethanol-fed rats;Carlen;Neurosci Lett,1980

4. Antagonism of the enhanced suseptibility to audiogenic seizures during alcohol withdrawal in the rat by GABA and GABA-mimetic agents;Cooper;J Pharmacol Exp Ther,1979

5. Suppression of ethanol-induced locomotor stimulation by GABA-like drugs;Cott;Naunyn Schmiedebergs Arch Pharmacol,1976

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