ET-1 increases reactive oxygen species following hypoxia and high-salt diet in the mouse glomerulus-Reference-Cited by-同舟云学术

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ET-1 increases reactive oxygen species following hypoxia and high-salt diet in the mouse glomerulus

Published:2014-09-29 Issue:3 Volume:213 Page:722-730
ISSN:1748-1708
Container-title:Acta Physiologica
language:en
Short-container-title:Acta Physiol

Author:

Heimlich J. B.¹,Speed J. S.²,Bloom C. J.³,O'Connor P. M.¹,Pollock J. S.²,Pollock D. M.²

Affiliation:

1. Department of Physiology; Georgia Regents University; Augusta GA USA

2. Cardio-Renal Physiology and Medicine; Division of Nephrology; University of Alabama at Birmingham; Birmingham AL USA

3. Department of Medicine; Georgia Regents University; Augusta GA USA

Funder

National Heart Lung and Blood Institute

Publisher

Wiley

Subject

Physiology

Reference24 articles.

1. Collecting duct-specific knockout of endothelin-1 causes hypertension and sodium retention;Ahn;J Clin Invest,2004

2. Interleukin-1beta, but not interleukin-6, enhances renal and systemic endothelin production in vivo;Boesen;Am J Physiol Renal Physiol,2008

3. ETA receptor blockade decreases vascular superoxide generation in DOCA-salt hypertension;Callera;Hypertension,2003

4. Preeclamptic sera induce nephrin shedding from podocytes through endothelin-1 release by endothelial glomerular cells;Collino;Am J Physiol Renal Physiol,2008

5. The endothelin system and its antagonism in chronic kidney disease;Dhaun;J Am Soc Nephrol,2006

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