Catecholaminergic polymorphic ventricular tachycardia, an update
Author:
Affiliation:
1. Design of Studies and Scientific Writing Laboratory in the ABC Medicine Faculty; Santo André; São Paulo Brazil
2. Coronary Center of the Messejana Hospital Dr. Carlos Alberto Studart Gomes; Fortaleza Ceará Brazil
Publisher
Wiley
Subject
Physiology (medical),Cardiology and Cardiovascular Medicine,General Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/anec.12512/fullpdf
Reference50 articles.
1. Distinct U wave changes in patients with catecholaminergic polymorphic ventricular tachycardia (CPVT);Aizawa;International Heart Journal,2006
2. The mechanism of flecainide action in CPVT does not involve a direct effect on RyR2;Bannister;Circulation Research,2015
3. Expanding spectrum of human RYR2-related disease: New electrocardiographic, structural, and genetic features;Bhuiyan;Circulation,2007
4. Exon 3 deletion of ryanodine receptor causes left ventricular noncompaction, worsening catecholaminergic polymorphic ventricular tachycardia, and sudden cardiac arrest;Campbell;American Journal of Medical Genetics. Part A,2015
5. Arrhythmogenic mechanisms in a mouse model of catecholaminergic polymorphic ventricular tachycardia;Cerrone;Circulation Research,2007
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