Lysophosphatidylcholine-induced cytotoxicity and protection by heparin in mouse brain bEND.3 endothelial cells-Reference-Cited by-同舟云学术

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Lysophosphatidylcholine-induced cytotoxicity and protection by heparin in mouse brain bEND.3 endothelial cells

Published:2018-08-05 Issue:1 Volume:33 Page:52-62
ISSN:0767-3981
Container-title:Fundamental & Clinical Pharmacology
language:en
Short-container-title:Fundam Clin Pharmacol

Author:

Tsai Tien-Yao¹²,Leong Iat-Lon³,Cheng Ka-Shun⁴⁵,Shiao Lian-Ru⁶,Su Tzu-Hui⁴,Wong Kar-Lok⁴,Chan Paul⁷,Leung Yuk-Man⁶^ORCID

Affiliation:

1. Cardiovascular Division; Fu Jen Catholic University Hospital; New Taipei City Taiwan

2. School of Medicine; College of Medicine; Fu Jen Catholic University; New Taipei City Taiwan

3. Division of Cardiology; Department of Internal Medicine; Kiang Wu Hospital; Macau China

4. Department of Anesthesiology; China Medical University Hospital; Taichung Taiwan

5. Department of Anesthesiology; The Qingdao University Yuhuangding Hospital; Yantai Shandong China

6. Department of Physiology; China Medical University; Taichung Taiwan

7. Division of Cardiology; Department of Medicine; Taipei Medical University Wan Fang Hospital; Taipei Taiwan

Funder

China Medical University Hospital

Ministry of Science and Technology, Taiwan

Publisher

Wiley

Subject

Pharmacology (medical),Pharmacology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/fcp.12399/fullpdf

Reference28 articles.

1. Atherosclerosis or lipoprotein-induced endothelial dysfunction. Potential mechanisms underlying reduction in EDRF/nitric oxide activity;Flavahan;Circulation,1992

2. Endothelial cell apoptosis: biochemical characteristics and potential implications for atherosclerosis;Choy;J. Mol. Cell. Cardiol.,2001

3. Impairment of endothelium-dependent arterial relaxation by lysolecithin in modified low-density lipoproteins;Kugiyama;Nature,1990

4. Oxidized low-density lipoprotein-induced apoptosis;Salvayre;Biochim. Biophys. Acta,2002

5. Oxidized low-density lipoprotein induces apoptotic insults to mouse cerebral endothelial cells via a Bax-mitochondria-caspase protease pathway;Chen;Toxicol. Appl. Pharmacol.,2007

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