Atherosclerosis or lipoprotein-induced endothelial dysfunction. Potential mechanisms underlying reduction in EDRF/nitric oxide activity.
Author:
Affiliation:
1. Department of Medicine, Johns Hopkins University School of Medicine, Baltimore, Md. 21205.
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Physiology (medical),Cardiology and Cardiovascular Medicine
Link
https://www.ahajournals.org/doi/pdf/10.1161/01.CIR.85.5.1927
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4. Nitric oxide-generating vasodilators and 8-bromo-cyclic guanosine monophosphate inhibit mitogenesis and proliferation of cultured rat vascular smooth muscle cells.
5. Mediators produced by the endothelial cell.
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