SETDB1: A perspective into immune cell function and cancer immunotherapy
Author:
Affiliation:
1. Department of Pathology, Carver College of Medicine University of Iowa Iowa City Iowa USA
2. Holden Comprehensive Cancer Center, Carver College of Medicine University of Iowa Iowa City Iowa USA
Funder
National Institutes of Health
Roy J. and Lucille A. Carver College of Medicine, University of Iowa
Publisher
Wiley
Subject
Immunology,Immunology and Allergy
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/imm.13619
Reference39 articles.
1. SETDB1 promotes the progression of colorectal cancer via epigenetically silencing p21 expression
2. SETDB1 Inhibits p53-Mediated Apoptosis and Is Required for Formation of Pancreatic Ductal Adenocarcinomas in Mice
3. SETDB1 is required for intestinal epithelial differentiation and the prevention of intestinal inflammation
4. SETDB1 induces epithelialmesenchymal transition in breast carcinoma by directly binding with snail promoter;Yang W;Oncol Rep,2019
5. Histone methyltransferase SETDB1 regulates liver cancer cell growth through methylation of p53
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3. SETDB1 promotes progression through upregulation of SF3B4 expression and regulates the immunity in ovarian cancer;Journal of Ovarian Research;2024-02-05
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