Modulation of Contractility in Failing Human Myocytes by Reverse-Mode Na/Ca Exchange-Reference-Cited by-同舟云学术

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Modulation of Contractility in Failing Human Myocytes by Reverse-Mode Na/Ca Exchange

Published:2006-01-24 Issue:1 Volume:976 Page:466-471
ISSN:0077-8923
Container-title:Annals of the New York Academy of Sciences
language:en
Short-container-title:

Author:

PIACENTINO VALENTINO,WEBER CHRISTOPHER R.,GAUGHAN JOHN P.,MARGULIES KENNETH B.,BERS DONALD M.,HOUSER STEVEN R.

Publisher

Wiley

Subject

History and Philosophy of Science,General Biochemistry, Genetics and Molecular Biology,General Neuroscience

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1749-6632.2002.tb04776.x/fullpdf

Reference16 articles.

1. Abnormalities of calcium cycling in the hypertrophied and failing heart;Houser;J. Mol. Cell. Cardiol.,2000

2. Ionic mechanism of action potential prolongation in ventricular myocytes from dogs with pacing-induced heart failure;Kääb;Circ. Res.,1996

3. Reduced contraction and altered frequency response of isolated ventricular myocytes from patients with heart failure;Davies;Circulation,1995

4. Alterations of sarcoplasmic reticulum proteins in failing human dilated cardiomyopathy;Meyer;Circulation,1995

5. Unchanged protein levels of SERCA II and phospholamban but reduced Ca2+ uptake and Ca2+-ATPase activity of cardiac sarcoplasmic reticulum from dilated cardiomyopathy patients compared with patients with nonfailing hearts;Schwinger;Circulation,1995

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2. Role of RyR2 Phosphorylation in Heart Failure and Arrhythmias;Circulation Research;2014-04-11

3. Targeted ablation of the histidine-rich Ca2+-binding protein (HRC) gene is associated with abnormal SR Ca2+-cycling and severe pathology under pressure-overload stress;Basic Research in Cardiology;2013-04-04

4. Overexpression of NaV1.6 channels is associated with the invasion capacity of human cervical cancer;International Journal of Cancer;2011-08-12

5. Dofetilide Enhances the Contractility of Rat Ventricular Myocytes via Augmentation of Na+–Ca2+ Exchange;Cardiovascular Drugs and Therapy;2009-02-17

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