Exploiting PI3K/mTOR signaling to accelerate epithelial wound healing-Reference-Cited by-同舟云学术

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Exploiting PI3K/mTOR signaling to accelerate epithelial wound healing

Published:2013-02-04 Issue:6 Volume:19 Page:551-558
ISSN:1354-523X
Container-title:Oral Diseases
language:en
Short-container-title:Oral Dis

Author:

Castilho RM¹,Squarize CH¹,Gutkind JS²

Affiliation:

1. Laboratory of Epithelial Biology, Department of Periodontics and Oral Medicine; University of Michigan School of Dentistry; Ann Arbor; MI; USA

2. Oral and Pharyngeal Cancer Branch; National Institute of Dental and Craniofacial Research, National Institutes of Health; Bethesda; MD; USA

Publisher

Wiley

Subject

General Dentistry,Otorhinolaryngology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/odi.12070/fullpdf

Reference56 articles.

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3. Periostin potently promotes metastatic growth of colon cancer by augmenting cell survival via the Akt/PKB pathway;Bao;Cancer Cell,2004

4. Phosphoinositide 3-kinase signaling to Akt promotes keratinocyte differentiation versus death;Calautti;J Biol Chem,2005

5. New insights into tumor suppression: PTEN suppresses tumor formation by restraining the phosphoinositide 3-kinase/AKT pathway;Cantley;Proc Natl Acad Sci USA,1999

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