Neurobiology of comorbid post-traumatic stress disorder and alcohol-use disorder-Reference-Cited by-同舟云学术

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Neurobiology of comorbid post-traumatic stress disorder and alcohol-use disorder

Published:2016-11-18 Issue:1 Volume:16 Page:15-43
ISSN:1601-1848
Container-title:Genes, Brain and Behavior
language:en
Short-container-title:

Author:

Gilpin N. W.¹²,Weiner J. L.³

Affiliation:

1. Department of Physiology; Louisiana State University Health Sciences Center; New Orleans LA

2. Neuroscience Center of Excellence; Louisiana State University Health Sciences Center; New Orleans LA

3. Department of Physiology and Pharmacology; Wake Forest School of Medicine; Winston-Salem NC USA

Funder

National Institutes of Health

Publisher

Wiley

Subject

Behavioral Neuroscience,Neurology,Genetics

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/gbb.12349/fullpdf

Reference396 articles.

1. Intravenous ethanol increases dopamine release in the ventral striatum in humans: PET study using bolus-plus-infusion administration of [(11)C]raclopride;Aalto;J Cereb Blood Flow Metab,2015

2. Imbalanced neural responsivity to risk and reward indicates stress vulnerability in humans;Admon;Cereb Cortex,2013a

3. A causal model of post-traumatic stress disorder: disentangling predisposed from acquired neural abnormalities;Admon;Trends Cogn Sci,2013b

4. Effect of early rearing conditions on alcohol drinking and 5-HT1A receptor function in C57BL/6J mice;Advani;Int J Neuropsychopharmacol,2007

5. Hippocampal volume in patients with alcohol dependence;Agartz;Arch Gen Psychiatry,1999

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2. Elevated GABAergic neurotransmission prevents chronic intermittent ethanol induced hyperexcitability of intrinsic and extrinsic inputs to the ventral subiculum of female rats;Neurobiology of Stress;2024-09

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