Elevated GABAergic neurotransmission prevents chronic intermittent ethanol induced hyperexcitability of intrinsic and extrinsic inputs to the ventral subiculum of female rats
Author:
Funder
National Institutes of Health
Publisher
Elsevier BV
Reference44 articles.
1. Enhanced ventral hippocampal synaptic transmission and impaired synaptic plasticity in a rodent model of alcohol addiction vulnerability;Almonte;Sci. Rep.,2017
2. Control of parallel hippocampal output pathways by amygdalar long-range inhibition;AlSubaie;Elife,2021
3. Chronic intermittent ethanol promotes ventral subiculum hyperexcitability via increases in extrinsic basolateral amygdala input and local network activity;Bach;Sci. Rep.,2021
4. Chemogenetic inhibition of a monosynaptic projection from the basolateral amygdala to the ventral hippocampus selectively reduces appetitive, but not consummatory, alcohol drinking-related behaviours;Bach;Eur. J. Neurosci.,2023
5. Chronic ethanol exposures leads to a negative affective state in female rats that is accompanied by a paradoxical decrease in ventral Hippocampus excitability;Bach;Front. Neurosci.,2021
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