CXCR 4 attenuates cardiomyocytes mitochondrial dysfunction to resist ischaemia‐reperfusion injury
Author:
Affiliation:
1. Department of Pathology & Lab Medicine College of Medicine University of Cincinnati Cincinnati OH USA
2. Department of Pharmacology & Cell Biophysics College of Medicine University of Cincinnati Cincinnati OH USA
Funder
National Institutes of Health
Publisher
Wiley
Subject
Cell Biology,Molecular Medicine
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/jcmm.12554
Reference30 articles.
1. Myocardial Reperfusion Injury
2. Myocardial ischemia-reperfusion injury: a neglected therapeutic target
3. Postconditioning and protection from reperfusion injury: where do we stand? * Position Paper from the Working Group of Cellular Biology of the Heart of the European Society of Cardiology
4. Structures of the CXCR4 Chemokine GPCR with Small-Molecule and Cyclic Peptide Antagonists
5. Up-regulation of leukocyte CXCR4 expression by sulfatide: An L-selectin-dependent pathway on CD4+ T cells
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