Cell-permeable peptide antioxidants as a novel therapeutic approach in a mouse model of amyotrophic lateral sclerosis-Reference-Cited by-同舟云学术

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Cell-permeable peptide antioxidants as a novel therapeutic approach in a mouse model of amyotrophic lateral sclerosis

Published:2006-08 Issue:4 Volume:98 Page:1141-1148
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J Neurochem

Author:

Petri Susanne,Kiaei Mahmoud,Damiano Maria,Hiller Andrew,Wille Elizabeth,Manfredi Giovanni,Calingasan Noel Y.,Szeto Hazel H.,Beal M. Flint

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1471-4159.2006.04018.x/fullpdf

Reference48 articles.

1. Increased oxidative damage to DNA in an animal model of amyotrophic lateral sclerosis

2. Mitochondria take center stage in aging and neurodegeneration

3. Increased 3-nitrotyrosine in both sporadic and familial amyotrophic lateral sclerosis

4. Elevated free nitrotyrosine levels, but not protein-bound nitrotyrosine or hydroxyl radicals, throughout amyotrophic lateral sclerosis (ALS)-like disease implicate tyrosine nitration as an aberrant in vivo property of one familial ALS-linked superoxide dismutase 1 mutant

5. Expression of a Cu,Zn superoxide dismutase typical of familial amyotrophic lateral sclerosis induces mitochondrial alteration and increase of cytosolic Ca2+concentration in transfected neuroblastoma SH-SY5Y cells

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