Susceptibility of porcine pulmonary microvascular endothelial cells to porcine reproductive and respiratory syndrome virus
Author:
Affiliation:
1. Beijing Key Laboratory of Traditional Chinese Veterinary Medicine, Beijing University of Agriculture, Beijing, 102206, P.R. China
2. Beijing Neurosurgical Institute, Beijing Tiantan Hospital, Beijing, 100000, P.R. China
Publisher
Japanese Society of Veterinary Science
Subject
General Veterinary
Link
https://www.jstage.jst.go.jp/article/jvms/82/9/82_20-0324/_pdf
Reference24 articles.
1. 1. Aird, W. C. 2012. Endothelial cell heterogeneity. Cold Spring Harb. Perspect. Med. 2: a006429.
2. 2. Dalrymple, N. A. and Mackow, E. R. 2014. Virus interactions with endothelial cell receptors: implications for viral pathogenesis. Curr. Opin. Virol. 7: 134–140.
3. 3. Dave, J. M. and Bayless, K. J. 2014. Vimentin as an integral regulator of cell adhesion and endothelial sprouting. Microcirculation 21: 333–344.
4. 4. Feng, L., Zhang, X., Xia, X., Li, Y., He, S. and Sun, H. 2013. Generation and characterization of a porcine endometrial endothelial cell line susceptible to porcine reproductive and respiratory syndrome virus. Virus Res. 171: 209–215.
5. 5. Gray, D. K., Dvorak, C. M. T., Robinson, S. R. and Murtaugh, M. P. 2019. Characterization of age-related susceptibility of macrophages to porcine reproductive and respiratory syndrome virus. Virus Res. 263: 139–144.
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