ICBP90 Expression Is Downregulated in Apoptosis-Induced Jurkat Cells

Author:

ABBADY ABDUL-QADER,BRONNER CHRISTIAN,TROTZIER MARIE-ALINE,HOPFNER RAPHAEL,BATHAMI KAWTAR,MULLER CHRISTIAN D.,JEANBLANC MICHAEL,MOUSLI MARC

Publisher

Wiley

Subject

History and Philosophy of Science,General Biochemistry, Genetics and Molecular Biology,General Neuroscience

Reference5 articles.

1. Apoptosis in cancer: cause and cure;Kaufmann;Bioessays,2000

2. ICBP90, a novel human CCAAT binding protein, involved in the regulation of topoisomerase IIa expression;Hopfner;Cancer Res.,2000

3. ICBP90 belongs to a new family of proteins with an expression that is deregulated in cancer cells;Mousli;Br. J. Cancer,2003

4. Overexpression of ICBP90, a novel CCAAT-binding protein, overcomes cell contact inhibition by forcingtopoisomerase IIa expression;Hopfner;Anticancer Res.,2002

5. Np95 is regulated by E1A during mitotic reactivation of terminally differentiated cells and is essential for S phase entry;Bonapace;J. Cell Biol.,2002

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