A Mouse Model of Classical Late-Infantile Neuronal Ceroid Lipofuscinosis Based on Targeted Disruption of the CLN2 Gene Results in a Loss of Tripeptidyl-Peptidase I Activity and Progressive Neurodegeneration-Reference-Cited by-同舟云学术

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A Mouse Model of Classical Late-Infantile Neuronal Ceroid Lipofuscinosis Based on Targeted Disruption of the CLN2 Gene Results in a Loss of Tripeptidyl-Peptidase I Activity and Progressive Neurodegeneration

Published:2004-10-13 Issue:41 Volume:24 Page:9117-9126
ISSN:0270-6474
Container-title:Journal of Neuroscience
language:en
Short-container-title:Journal of Neuroscience

Author:

Sleat D. E.

Publisher

Society for Neuroscience

Subject

General Neuroscience

Cited by 126 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. GABAergic interneurons contribute to the fatal seizure phenotype of CLN2 disease mice;2024-03-30

2. The involvement of Purkinje cells in progressive myoclonic epilepsy: Focus on neuronal ceroid lipofuscinosis;Neurobiology of Disease;2023-09

3. Gene therapy ameliorates spontaneous seizures associated with cortical neuron loss in a Cln2R207X mouse model;Journal of Clinical Investigation;2023-06-15

4. Extracellular Vesicles Released by Genetically Modified Macrophages Activate Autophagy and Produce Potent Neuroprotection in Mouse Model of Lysosomal Storage Disorder, Batten Disease;Cells;2023-05-29

5. Cynomolgus macaque model of neuronal ceroid lipofuscinosis type 2 disease;Experimental Neurology;2023-05

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