Caspase-6 Activity in a BACHD Mouse Modulates Steady-State Levels of Mutant Huntingtin Protein But Is Not Necessary for Production of a 586 Amino Acid Proteolytic Fragment-Reference-Cited by-同舟云学术

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Caspase-6 Activity in a BACHD Mouse Modulates Steady-State Levels of Mutant Huntingtin Protein But Is Not Necessary for Production of a 586 Amino Acid Proteolytic Fragment

Published:2012-05-30 Issue:22 Volume:32 Page:7454-7465
ISSN:0270-6474
Container-title:Journal of Neuroscience
language:en
Short-container-title:Journal of Neuroscience

Author:

Gafni J.,Papanikolaou T.,DeGiacomo F.,Holcomb J.,Chen S.,Menalled L.,Kudwa A.,Fitzpatrick J.,Miller S.,Ramboz S.,Tuunanen P. I.,Lehtimaki K. K.,Yang X. W.,Park L.,Kwak S.,Howland D.,Park H.,Ellerby L. M.

Publisher

Society for Neuroscience

Subject

General Neuroscience

Cited by 44 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

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3. Inhibiting cellular uptake of mutant huntingtin using a monoclonal antibody: Implications for the treatment of Huntington's disease;Neurobiology of Disease;2020-07

4. Truncation of mutant huntingtin in knock-in mice demonstrates exon1 huntingtin is a key pathogenic form;Nature Communications;2020-05-22

5. The Inverse Comorbidity between Oncological Diseases and Huntington’s Disease: Review of Epidemiological and Biological Evidence;Russian Journal of Genetics;2020-03

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