The Inverse Comorbidity between Oncological Diseases and Huntington’s Disease: Review of Epidemiological and Biological Evidence
Author:
Publisher
Pleiades Publishing Ltd
Subject
Genetics
Link
http://link.springer.com/content/pdf/10.1134/S1022795420030059.pdf
Reference124 articles.
1. Bates, G.P., Dorsey, R., Gusella, J.F., et al., Huntington disease, Nat. Rev. Dis. Primers, 2015, vol. 1, p. 15005. https://doi.org/10.1038/nrdp.2015.5
2. Reiner, A., Albin, R.L., Anderson, K.D., et al., Differential loss of striatal projection neurons in Huntington disease, Proc. Natl. Acad. Sci. U.S.A., 1988, vol. 85, pp. 5733—5737.
3. The Huntington’s Disease Collaborative Research Group, A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington’s disease chromosomes, Cell, 1993, vol. 72, pp. 971—983. https://doi.org/10.1016/00928674(93)90585-E
4. Kremer, B., Goldberg, P., Andrew, S.E., et al., A worldwide study of the Huntington’s disease mutation: the sensitivity and specificity of measuring CAG repeats, N. Engl. J. Med., 1994, vol. 330, pp. 1401—1406. https://doi.org/10.1056/NEJM199405193302001
5. Ha, A.D. and Jankovic, J., Exploring the correlates of intermediate CAG repeats in Huntington disease, Postgrad. Med. J., 2011, vol. 123, no. 5, pp. 116—121. https://doi.org/10.3810/pgm.2011.09.2466
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