Genetic Mapping of APP and Amyloid-β Biology Modulation by Trisomy 21-Reference-Cited by-同舟云学术

Genetic Mapping of APP and Amyloid-β Biology Modulation by Trisomy 21

Published:2022-07-14 Issue:33 Volume:42 Page:6453-6468
ISSN:0270-6474
Container-title:The Journal of Neuroscience
language:en
Short-container-title:J. Neurosci.

Author:

Mumford Paige^ORCID,Tosh Justin,Anderle Silvia,Gkanatsiou Wikberg Eleni,Lau Gloria,Noy Sue,Cleverley Karen,Saito Takashi^ORCID,Saido Takaomi C.^ORCID,Yu Eugene,Brinkmalm Gunnar,Portelius Erik,Blennow Kaj,Zetterberg Henrik,Tybulewicz Victor^ORCID,Fisher Elizabeth M.C.,Wiseman Frances K.

Publisher

Society for Neuroscience

Subject

General Neuroscience

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1. Amyloid-β peptide signature associated with cerebral amyloid angiopathy in familial Alzheimer’s disease with APPdup and Down syndrome;Acta Neuropathologica;2024-07-18

2. Reduction of Cystatin B results in increased cathepsin B activity in disomic but not Trisomy21 human cellular and mouse models;2024-04-14

3. Topical Cellular/Tissue and Molecular Aspects Regarding Nonpharmacological Interventions in Alzheimer’s Disease—A Systematic Review;International Journal of Molecular Sciences;2023-11-20

4. Cathepsin B abundance, activity and microglial localisation in Alzheimer’s disease-Down syndrome and early onset Alzheimer’s disease; the role of elevated cystatin B;Acta Neuropathologica Communications;2023-08-14

5. Cathepsin B abundance, activity and microglial localisation in Alzheimer’s disease – Down syndrome and early onset Alzheimer’s disease; the role of elevated cystatin B;2023-06-23