p38MAPK Signaling and Desmoglein-3 Internalization Are Linked Events in Pemphigus Acantholysis
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference25 articles.
1. Ultrastructural Studies of Acantholysis Induced In Vivo by Passive Transfer of IgG from Endemic Pemphigus Foliaceus (Fogo Selvagem)
2. Pemphigus Vulgaris-IgG Causes a Rapid Depletion of Desmoglein 3 (Dsg3) from the Triton X-100 Soluble Pools, Leading to the Formation of Dsg3-Depleted Desmosomes in a Human Squamous Carcinoma Cell Line, DJM-1 Cells
3. Desmoglein Endocytosis and Desmosome Disassembly Are Coordinated Responses to Pemphigus Autoantibodies
4. Changes in desmoglein 1 expression and subcellular localization in cultured keratinocytes subjected to anti-desmoglein 1 pemphigus autoimmunity
5. Pemphigus Vulgaris IgG-induced Desmoglein-3 Endocytosis and Desmosomal Disassembly Are Mediated by a Clathrin- and Dynamin-independent Mechanism
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