Hyperglycemia-induced inflamm-aging accelerates gingival senescence via NLRC4 phosphorylation-Reference-Cited by-同舟云学术

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Hyperglycemia-induced inflamm-aging accelerates gingival senescence via NLRC4 phosphorylation

Published:2019-12 Issue:49 Volume:294 Page:18807-18819
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Zhang Peng,Wang Qian,Nie Lulingxiao,Zhu Rui,Zhou Xinyi,Zhao Pengfei,Ji Ning,Liang Xing,Ding Yi,Yuan Quan,Wang Qi^ORCID

Funder

NSF | Foundation for Innovative Research Groups of the National Natural Science Foundation of China

Internatioal Scientific Cooperation and Exanges Project of Sichuan Province

International Cooperation Project of Chengdu Municipal Science and Technology Bureau

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference56 articles.

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2. Diabetes mellitus and periodontitis: a tale of two common interrelated diseases;Lalla;Nat. Rev. Endocrinol,2011

3. The influence of type 1 and type 2 diabetes on periodontal disease progression: prospective results from the Study of Health in Pomerania (SHIP);Demmer;Diabetes Care,2012

4. “Inflammaging” as a druggable target: a senescence-associated secretory phenotype-centered view of type 2 diabetes;Prattichizzo;Oxid Med. Cell Longev,2016

5. The number of p16INK4a positive cells in human skin reflects biological age;Waaijer;Aging Cell,2012

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2. Pathology of Diabetes-Induced Immune Dysfunction;International Journal of Molecular Sciences;2024-06-28

3. Molecular Signatures of Senescence in Periodontitis: Clinical Insights;Journal of Dental Research;2024-06-14

4. Intermittent hyperglycaemia induces macrophage dysfunction by extracellular regulated protein kinase‐dependent PKM2 translocation in periodontitis;Cell Proliferation;2024-05-24

5. The senescence-associated secretory phenotype and its physiological and pathological implications;Nature Reviews Molecular Cell Biology;2024-04-23

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