In Situ Ca2+ Dependence for Activation of Ca2+/Calmodulin- dependent Protein Kinase II in Vascular Smooth Muscle Cells-Reference-Cited by-同舟云学术

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In Situ Ca2+ Dependence for Activation of Ca2+/Calmodulin- dependent Protein Kinase II in Vascular Smooth Muscle Cells

Published:1996-02 Issue:5 Volume:271 Page:2506-2513
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Abraham S. Thomas,Benscoter Holly,Schworer Charles M.,Singer Harold A.

Funder

National Institutes of Health

American Heart Association (Pennsylvania affiliate) Fellowship

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference45 articles.

1. Down-regulation of protein kinase C potentiates angiotensin II-stimulated polyphosphoinositide hydrolysis in vascular smooth-muscle cells

2. Signal transduction and regulation in smooth muscle

3. Activation of MAP kinases by calcium-dependent and calcium-independent pathways. Stimulation by thapsigargin and epidermal growth factor.

4. Regulation of Gene Expression in Hippocampal Neurons by Distinct Calcium Signaling Pathways

5. Intracellular free Ca2+ in the cell cycle in human fibroblasts: transitions between G1 and G0 and progression into S phase.

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1. A non-radioactive in vitro CaMKII activity assay using HPLC-MS;Journal of Pharmacological and Toxicological Methods;2018-11

2. Combined mechanisms of neural firing rate homeostasis;Biological Cybernetics;2018-06-28

3. Ca2+/Calmodulin-Dependent Protein Kinase II in Vascular Smooth Muscle;Advances in Pharmacology;2017

4. Role of CaMKII in Ang-II-dependent small artery remodeling;Vascular Pharmacology;2016-12

5. Ca 2+ /calmodulin‐dependent protein kinase II‐γ (CaMKIIγ) negatively regulates vascular smooth muscle cell proliferation and vascular remodeling;The FASEB Journal;2015-11-13

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