Ca 2+ /calmodulin‐dependent protein kinase II‐γ (CaMKIIγ) negatively regulates vascular smooth muscle cell proliferation and vascular remodeling
Author:
Affiliation:
1. Center for Cardiovascular SciencesAlbany Medical CollegeAlbanyNew YorkUSA
2. Department of Cardiology, Angiology and PneumologyUniversity of HeidelbergHeidelbergGermany
Funder
National Institutes of Health
Publisher
Wiley
Subject
Genetics,Molecular Biology,Biochemistry,Biotechnology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.15-279158
Reference68 articles.
1. Molecular Determinants of Vascular Smooth Muscle Cell Diversity
2. Ca2+/calmodulin-dependent protein kinase II function in vascular remodelling
3. Excitation–Transcription Coupling in Arterial Smooth Muscle
4. Targeting of a Novel Ca+ 2 /Calmodulin-Dependent Protein Kinase II Is Essential for Extracellular Signal-Regulated Kinase–Mediated Signaling in Differentiated Smooth Muscle Cells
5. Inhibition of CaM kinase II activation and force maintenance by KN-93 in arterial smooth muscle
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