Alzheimer's Disease βA4 Protein Release and Amyloid Precursor Protein Sorting Are Regulated by Alternative Splicing-Reference-Cited by-同舟云学术

Alzheimer's Disease βA4 Protein Release and Amyloid Precursor Protein Sorting Are Regulated by Alternative Splicing

Published:1996-05 Issue:22 Volume:271 Page:13208-13214
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Hartmann Tobias,Bergsdorf Christian,Sandbrink Rupert,Tienari Pentti J.,Multhaup Gerd,Ida Nobuo,Bieger Sophie,Dyrks Thomas,Weidemann Andreas,Masters Colin L.,Beyreuther Konrad

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference33 articles.

1. The precursor of Alzheimer's disease amyloid A4 protein resembles a cell-surface receptor

2. Identification, biogenesis, and localization of precursors of Alzheimer's disease A4 amyloid protein

3. Identification, transmembrane orientation and biogenesis of the amyloid A4 precursor of Alzheimer's disease.

4. Beta A4-amyloid protein precursor mRNA isoforms without exon 15 are ubiquitously expressed in rat tissues including brain, but not in neurons.

5. Identification and differential expression of a novel alternative splice isoform of the beta A4 amyloid precursor protein (APP) mRNA in leukocytes and brain microglial cells.

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