Chronic Oxidative Stress as a Central Mechanism for Glucose Toxicity in Pancreatic Islet Beta Cells in Diabetes
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference59 articles.
1. Glucose autoxidation and protein modification. The potential role of ‘autoxidative glycosylation’ in diabetes
2. The pancreatic β-cell recognition of insulin secretagogues
3. Mitochondrial reactive oxygen species reduce insulin secretion by pancreatic β-cells
4. Inhibition of GAPDH activity by poly(ADP-ribose) polymerase activates three major pathways of hyperglycemic damage in endothelial cells
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