Selective Modulation of the Major Histocompatibility Complex Class II Antigen Presentation Pathway following B Cell Receptor Ligation and Protein Kinase C Activation-Reference-Cited by-同舟云学术

Selective Modulation of the Major Histocompatibility Complex Class II Antigen Presentation Pathway following B Cell Receptor Ligation and Protein Kinase C Activation

Published:1997-02 Issue:6 Volume:272 Page:3641-3647
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Barois Nicolas,Forquet Frédérique,Davoust Jean

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference62 articles.

1. MHC-dependent antigen processing and peptide presentation: Providing ligands for T lymphocyte activation

2. Assembly, Transport, and Function of MHC Class II Molecules

3. The segment of invariant chain that is critical for association with major histocompatibility complex class II molecules contains the sequence of a peptide eluted from class II polypeptides.

4. The CLIP region of invariant chain plays a critical role in regulating major histocompatibility complex class II folding, transport, and peptide occupancy.

Cited by 34 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. B-cell receptor dependent phagocytosis and presentation of particulate antigen by chronic lymphocytic leukemia cells;Exploration of Targeted Anti-tumor Therapy;2022-02-25

2. The Intramembrane Proteases Signal Peptide Peptidase-Like 2a and 2b Have Distinct Functions In Vivo;Molecular and Cellular Biology;2014-04-15

3. The intramembrane protease SPPL2a promotes B cell development and controls endosomal traffic by cleavage of the invariant chain;Journal of Experimental Medicine;2012-12-24

4. Dynamics of dendritic cell-derived vesicles: high-resolution flow cytometric analysis of extracellular vesicle quantity and quality;Journal of Leukocyte Biology;2012-12-17

5. Endosomally Stored MHC Class II Does Not Contribute to Antigen Presentation by Dendritic Cells at Inflammatory Conditions;Traffic;2011-05-13