Blocking c-Jun N-terminal Kinase (JNK) Translocation to the Mitochondria Prevents 6-Hydroxydopamine-induced Toxicity in Vitro and in Vivo-Reference-Cited by-同舟云学术

Blocking c-Jun N-terminal Kinase (JNK) Translocation to the Mitochondria Prevents 6-Hydroxydopamine-induced Toxicity in Vitro and in Vivo

Published:2013-01 Issue:2 Volume:288 Page:1079-1087
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Chambers Jeremy W.,Howard Shannon,LoGrasso Philip V.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference23 articles.

1. Direct activation of mitochondrial apoptosis machinery by c-Jun N-terminal kinase in adult cardiac myocytes;Aoki;J. Biol. Chem.,2002

2. Role of JNK translocation to mitochondria leading to inhibition of mitochondria bioenergetics in acetaminophen-induced liver injury;Hanawa;J. Biol. Chem.,2008

3. Cerebral ischemia provokes a profound exchange of activated JNK isoforms in brain mitochondria;Zhao;Mol. Cell. Neurosci.,2009

4. c-Jun N-terminal kinase regulates mitochondrial bioenergetics by modulating pyruvate dehydrogenase activity in primary cortical neurons;Zhou;J. Neurochem.,2008

5. Activation of c-Jun N-terminal kinase and decline of mitochondrial pyruvate dehydrogenase activity during brain aging;Zhou;FEBS Lett.,2009

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