The Effects of Mutations in Helices 4 and 6 of ApoA-I on Scavenger Receptor Class B Type I (SR-BI)-mediated Cholesterol Efflux Suggest That Formation of a Productive Complex between Reconstituted High Density Lipoprotein and SR-BI Is Required for Efficient Lipid Transport-Reference-Cited by-同舟云学术

The Effects of Mutations in Helices 4 and 6 of ApoA-I on Scavenger Receptor Class B Type I (SR-BI)-mediated Cholesterol Efflux Suggest That Formation of a Productive Complex between Reconstituted High Density Lipoprotein and SR-BI Is Required for Efficient Lipid Transport

Published:2002-06 Issue:24 Volume:277 Page:21576-21584
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Liu Tong,Krieger Monty,Kan Horng-Yuan,Zannis Vassilis I.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference64 articles.

1. Mutations in ABC1 in Tangier disease and familial high-density lipoprotein deficiency

2. The gene encoding ATP-binding cassette transporter 1 is mutated in Tangier disease

3. Tangier disease is caused by mutations in the gene encoding ATP-binding cassette transporter 1

4. The Tangier disease gene product ABC1 controls the cellular apolipoprotein-mediated lipid removal pathway

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