Tyrosine Phosphorylation of IκBα Activates NFκB through a Redox-regulated and c-Src-dependent Mechanism Following Hypoxia/Reoxygenation
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference33 articles.
1. Redox-Mediated Gene Therapies for Environmental Injury: Approaches and Concepts
2. Role of Xanthine Oxidase and Its Inhibitor in Hypoxia: Reoxygenation Injury
3. Reactive oxygen intermediates as apparently widely used messengers in the activation of the NF-kappa B transcription factor and HIV-1.
4. Phosphorylation Meets Ubiquitination: The Control of NF-κB Activity
5. The IκB Kinase Complex (IKK) Contains Two Kinase Subunits, IKKα and IKKβ, Necessary for IκB Phosphorylation and NF-κB Activation
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