Response to Shilling et al. (10.1074/jbc.M111.300491)
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference5 articles.
1. Lack of evidence for presenilins as endoplasmic reticulum Ca2+ leak channels;Shilling;J. Biol. Chem.,2012
2. Capacitative calcium entry deficits and elevated luminal calcium content in mutant presenilin-1 knock-in mice;Leissring;J. Cell Biol.,2000
3. Presenilins form ER Ca2+ leak channels, a function disrupted by familial Alzheimer disease-linked mutations;Tu;Cell,2006
4. Role of presenilins in neuronal calcium homeostasis;Zhang;J. Neurosci.,2010
5. Enhanced ryanodine receptor recruitment contributes to Ca2+ disruptions in young, adult, and aged Alzheimer disease mice;Stutzmann;J. Neurosci.,2006
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