Presenilins Form ER Ca2+ Leak Channels, a Function Disrupted by Familial Alzheimer's Disease-Linked Mutations-Reference-Cited by-同舟云学术

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Presenilins Form ER Ca2+ Leak Channels, a Function Disrupted by Familial Alzheimer's Disease-Linked Mutations

Published:2006-09 Issue:5 Volume:126 Page:981-993
ISSN:0092-8674
Container-title:Cell
language:en
Short-container-title:Cell

Author:

Tu Huiping,Nelson Omar,Bezprozvanny Arseny,Wang Zhengnan,Lee Sheu-Fen,Hao Yi-Heng,Serneels Lutgarde,De Strooper Bart,Yu Gang,Bezprozvanny Ilya

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference41 articles.

1. Presenilin 1 controls gamma-secretase processing of amyloid precursor protein in pre-golgi compartments of hippocampal neurons;Annaert;J. Cell Biol.,1999

2. Alzheimer disease amyloid beta protein forms calcium channels in bilayer membranes: blockade by tromethamine and aluminum;Arispe;Proc. Natl. Acad. Sci. USA,1993

3. Altered calcium homeostasis and mitochondrial dysfunction in cortical synaptic compartments of presenilin-1 mutant mice;Begley;J. Neurochem.,1999

4. Calcium leak from intracellular stores–the enigma of calcium signalling;Camello;Cell Calcium,2002

5. The presenilin 1 deltaE9 mutation gives enhanced basal phospholipase C activity and a resultant increase in intracellular calcium concentrations;Cedazo-Minguez;J. Biol. Chem.,2002

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