N-terminal Truncation of Antiapoptotic MCL1, but Not G2/M-induced Phosphorylation, Is Associated with Stabilization and Abundant Expression in Tumor Cells-Reference-Cited by-同舟云学术

N-terminal Truncation of Antiapoptotic MCL1, but Not G2/M-induced Phosphorylation, Is Associated with Stabilization and Abundant Expression in Tumor Cells

Published:2007-08 Issue:33 Volume:282 Page:23919-23936
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

De Biasio Alfredo,Vrana Julie A.,Zhou Ping,Qian Liping,Bieszczad Christine K.,Braley Karen E.,Domina Aaron M.,Weintraub Steven J.,Neveu John M.,Lane William S.,Craig Ruth W.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference71 articles.

1. Obligate Role of Anti-Apoptotic MCL-1 in the Survival of Hematopoietic Stem Cells

2. Development and maintenance of B and T lymphocytes requires antiapoptotic MCL-1

3. Dynamic changes in Mcl-1 expression regulate macrophage viability or commitment to apoptosis during bacterial clearance

4. Constitutively Activated Akt-1 Is Vital for the Survival of Human Monocyte-Differentiated Macrophages

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