c-Jun N-terminal Kinase (JNK)-dependent Acute Liver Injury from Acetaminophen or Tumor Necrosis Factor (TNF) Requires Mitochondrial Sab Protein Expression in Mice-Reference-Cited by-同舟云学术

c-Jun N-terminal Kinase (JNK)-dependent Acute Liver Injury from Acetaminophen or Tumor Necrosis Factor (TNF) Requires Mitochondrial Sab Protein Expression in Mice

Published:2011-10 Issue:40 Volume:286 Page:35071-35078
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Win Sanda,Than Tin Aung,Han Derick,Petrovic Lydia M.,Kaplowitz Neil

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference39 articles.

1. The c-jun kinase/stress-activated pathway: Regulation, function and role in human disease

2. Cell Signaling in Oxidative Stress-Induced Liver Injury

3. Redox Regulation of Tumor Necrosis Factor Signaling

4. The JNK signal transduction pathway

5. NF-κB inhibition sensitizes hepatocytes to TNF-induced apoptosis through a sustained activation of JNK and c-Jun

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