Normal Development of Mice and Unimpaired Cell Adhesion/Cell Motility/Actin-based Cytoskeleton without Compensatory Up-regulation of Ezrin or Radixin in Moesin Gene Knockout-Reference-Cited by-同舟云学术

Normal Development of Mice and Unimpaired Cell Adhesion/Cell Motility/Actin-based Cytoskeleton without Compensatory Up-regulation of Ezrin or Radixin in Moesin Gene Knockout

Published:1999-01 Issue:4 Volume:274 Page:2315-2321
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Doi Yoshinori,Itoh Masahiko,Yonemura Shigenobu,Ishihara Satoru,Takano Hiroshi,Noda Tetsuo,Tsukita Shoichiro,Tsukita Sachiko

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference63 articles.

1. Purification of an 80,000-dalton protein that is a component of the isolated microvillus cytoskeleton, and its localization in nonmuscle cells.

2. Microvillus-specific Mr 75,000 plasma membrane protein of human choriocarcinoma cells.

3. Rapid phosphorylation and reorganization of ezrin and spectrin accompany morphological changes induced in A-431 cells by epidermal growth factor.

4. The protein-tyrosine kinase substrate, p81, is homologous to a chicken microvillar core protein.

5. Epidermal growth factor induces rapid tyrosine phosphorylation of proteins in A431 human tumor cells

Cited by 139 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Structural Basis for the Interaction between the Ezrin FERM-Domain and Human Aquaporins;International Journal of Molecular Sciences;2024-07-12

2. Moesin deficiency leads to lupus-like nephritis with accumulation of CXCL13-producing patrolling monocytes;Biochemical and Biophysical Research Communications;2024-06

3. Comprehensive Genetic Profiling Reveals Frequent Alterations of Driver Genes on the X Chromosome in Extranodal NK/T-cell Lymphoma;Cancer Research;2024-04-24

4. ARHGAP18-ezrin functions as an autoregulatory module for RhoA in the assembly of distinct actin-based structures;eLife;2024-01-09

5. Two cases of pediatric primary immunodeficiency caused by a familial moesin(MSN)gene mutation;Clinical Immunology;2024-01