Insulin Receptor Substrate-2 Is Not Necessary for Insulin- and Exercise-stimulated Glucose Transport in Skeletal Muscle-Reference-Cited by-同舟云学术

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Insulin Receptor Substrate-2 Is Not Necessary for Insulin- and Exercise-stimulated Glucose Transport in Skeletal Muscle

Published:1999-07 Issue:30 Volume:274 Page:20791-20795
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Higaki Yasuki,Wojtaszewski Jørgen F.P.,Hirshman Michael F.,Withers Dominic J.,Towery Heather,White Morris F.,Goodyear Laurie J.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference31 articles.

1. The Triumvirate: β-Cell, Muscle, Liver: A Collusion Responsible for NIDDM

2. Regulation of insulin receptor substrate-1 in liver and muscle of animal models of insulin resistance.

3. Insulin receptor phosphorylation, insulin receptor substrate-1 phosphorylation, and phosphatidylinositol 3-kinase activity are decreased in intact skeletal muscle strips from obese subjects.

4. Defect in skeletal muscle phosphatidylinositol-3-kinase in obese insulin-resistant mice.

5. Insulin Receptor Substrate-1 Phosphorylation and Phosphatidylinositol 3-Kinase Activity in Skeletal Muscle From NIDDM Subjects After In Vivo Insulin Stimulation

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