Overexpression of β2-Adrenergic Receptors cAMP-dependent Protein Kinase Phosphorylates and Modulates Slow Delayed Rectifier Potassium Channels Expressed in Murine Heart-Reference-Cited by-同舟云学术

Overexpression of β2-Adrenergic Receptors cAMP-dependent Protein Kinase Phosphorylates and Modulates Slow Delayed Rectifier Potassium Channels Expressed in Murine Heart

Published:2004-09 Issue:39 Volume:279 Page:40778-40787
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Dilly Keith W.,Kurokawa Junko,Terrenoire Cecile,Reiken Steven,Lederer W.J.,Marks Andrew R.,Kass Robert S.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference29 articles.

1. Molecular physiology of cardiac potassium channels

2. Coassembly of KVLQT1 and minK (IsK) proteins to form cardiac IKS potassium channel

3. KvLQT1 and IsK (minK) proteins associate to form the IKS cardiac potassium current

4. Properties and regulation of the minK potassium channel protein

5. Probing the Contribution of I Ks to Canine Ventricular Repolarization

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