Characterization and functional role of rapid- and slow-activating delayed rectifier K+ currents in atrioventricular node cells of guinea pigs
Author:
Publisher
Springer Science and Business Media LLC
Subject
Physiology (medical),Clinical Biochemistry,Physiology
Link
https://link.springer.com/content/pdf/10.1007/s00424-021-02617-z.pdf
Reference42 articles.
1. Andrew Atkinson HD (2015) Molecular investigation into the human atrioventricular node in heart failure. Anat Physiol 05. https://doi.org/10.4172/2161-0940.1000164
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3. Banyasz T, Jian Z, Horvath B, Khabbaz S, Izu LT, Chen-Izu Y (2014) Beta-adrenergic stimulation reverses the I Kr-I Ks dominant pattern during cardiac action potential. Pflugers Arch - Eur J Physiol 466:2067–2076. https://doi.org/10.1007/s00424-014-1465-7
4. Bénitah JP, Gomez AM, Bailly P, Da Ponte JP, Berson G, Delgado C, Lorente P (1993) Heterogeneity of the early outward current in ventricular cells isolated from normal and hypertrophied rat hearts. J Physiol 469:111–138. https://doi.org/10.1113/jphysiol.1993.sp019807
5. Bosch RF, Gaspo R, Busch AE, Lang HJ, Li GR, Nattel S (1998) Effects of the chromanol 293B, a selective blocker of the slow, component of the delayed rectifier K+ current, on repolarization in human and guinea pig ventricular myocytes. Cardiovasc Res 38:441–450. https://doi.org/10.1016/s0008-6363(98)00021-2
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