Regulation of Steady-state β-Amyloid Levels in the Brain by Neprilysin and Endothelin-converting Enzyme but Not Angiotensin-converting Enzyme-Reference-Cited by-同舟云学术

Regulation of Steady-state β-Amyloid Levels in the Brain by Neprilysin and Endothelin-converting Enzyme but Not Angiotensin-converting Enzyme

Published:2006-10 Issue:41 Volume:281 Page:30471-30478
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Eckman Elizabeth A.,Adams Stephanie K.,Troendle Frederick J.,Stodola Becky A.,Kahn Murad A.,Fauq Abdul H.,Xiao Hong D.,Bernstein Kenneth E.,Eckman Christopher B.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference49 articles.

1. The Aβ Hypothesis: Leading Us to Rationally-Designed Therapeutic Strategies for the Treatment or Prevention of Alzheimer Disease

2. Expanding the association between theAPOEgene and the risk of Alzheimer's disease: possible roles forAPOEpromoter polymorphisms and alterations inAPOEtranscription

3. Aβ-degrading enzymes: modulators of Alzheimer's disease pathogenesis and targets for therapeutic intervention

4. Metabolic Regulation of Brain Aβ by Neprilysin

5. Alzheimer's Disease β-Amyloid Peptide Is Increased in Mice Deficient in Endothelin-converting Enzyme

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