TAK1 Is Recruited to the Tumor Necrosis Factor-α (TNF-α) Receptor 1 Complex in a Receptor-interacting Protein (RIP)-dependent Manner and Cooperates with MEKK3 Leading to NF-κB Activation-Reference-Cited by-同舟云学术

TAK1 Is Recruited to the Tumor Necrosis Factor-α (TNF-α) Receptor 1 Complex in a Receptor-interacting Protein (RIP)-dependent Manner and Cooperates with MEKK3 Leading to NF-κB Activation

Published:2005-12 Issue:52 Volume:280 Page:43056-43063
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Blonska Marzenna,Shambharkar Prashant B.,Kobayashi Masayuki,Zhang Dongyu,Sakurai Hiroaki,Su Bing,Lin Xin

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference34 articles.

1. Mitogen-activated protein kinase cascades and regulation of gene expression

2. MEK Kinase 3 Directly Activates MKK6 and MKK7, Specific Activators of the p38 and c-Jun NH2-terminal Kinases

3. TAK1 is a ubiquitin-dependent kinase of MKK and IKK

4. TGF-β-Activated Kinase 1 Stimulates NF-κB Activation by an NF-κB-Inducing Kinase-Independent Mechanism

5. Mitogen-activated Protein Kinase/ERK Kinase Kinases 2 and 3 Activate Nuclear Factor-κB through IκB Kinase-α and IκB Kinase-β

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