Tafazzin deficiency impairs CoA-dependent oxidative metabolism in cardiac mitochondria-Reference-Cited by-同舟云学术

Tafazzin deficiency impairs CoA-dependent oxidative metabolism in cardiac mitochondria

Published:2020-08 Issue:35 Volume:295 Page:12485-12497
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Le Catherine H.,Benage Lindsay G.,Specht Kalyn S.,Li Puma Lance C.,Mulligan Christopher M.,Heuberger Adam L.,Prenni Jessica E.,Claypool Steven M.^ORCID,Chatfield Kathryn C.^ORCID,Sparagna Genevieve C.,Chicco Adam J.

Funder

Barth Syndrome Foundation

American Heart Association

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference69 articles.

1. A novel X-linked gene, G4.5. is responsible for Barth syndrome;Bione;Nat. Genet,1996

2. X-linked cardioskeletal myopathy and neutropenia (Barth syndrome) (MIM 302060);Barth;J. Inherit. Metab. Dis,1999

3. Cardiolipins and biomembrane function;Hoch;Biochim. Biophys. Acta,1992

4. Cardiolipin requirement for electron transfer in complex I and III of the mitochondrial respiratory chain;Fry;J. Biol. Chem,1981

5. Functional binding of cardiolipin to cytochrome c oxidase;Robinson;J. Bioenerg. Biomembr,1993

Cited by 26 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. The subtherapeutic dose of valproic acid induces the activity of cardiolipin-dependent proteins;Biochimica et Biophysica Acta (BBA) - Bioenergetics;2024-11

2. Dilated Cardiomyopathy in Children;Pediatric Cardiology;2024

3. Activation of the integrated stress response rewires cardiac metabolism in Barth syndrome;Basic Research in Cardiology;2023-11-06

4. Elevated liver glycogenolysis mediates higher blood glucose during acute exercise in Barth syndrome;PLOS ONE;2023-08-31

5. Metabolic switch from fatty acid oxidation to glycolysis in knock‐in mouse model of Barth syndrome;EMBO Molecular Medicine;2023-08-03