Phenotypic Spectrum Caused by Transgenic Overexpression of Activated Akt in the Heart-Reference-Cited by-同舟云学术

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Phenotypic Spectrum Caused by Transgenic Overexpression of Activated Akt in the Heart

Published:2002-06 Issue:25 Volume:277 Page:22896-22901
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Matsui Takashi,Li Ling,Wu Justina C.,Cook Stuart A.,Nagoshi Tomohisa,Picard Michael H.,Liao Ronglih,Rosenzweig Anthony

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference49 articles.

1. Physiological Role of Akt in Insulin-Stimulated Translocation of GLUT4 in Transfected Rat Adipose Cells

2. Myocardial protection by insulin is dependent on phospatidylinositol 3-kinase but not protein kinase C or K ATP channels in the isolated rabbit heart

3. Antiapoptotic signalling by the insulin-like growth factor I receptor, phosphatidylinositol 3-kinase, and Akt

4. Regulation of Neuronal Survival by the Serine-Threonine Protein Kinase Akt

5. Cardioprotective effect of insulin-like growth factor I in myocardial ischemia followed by reperfusion.

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