A Role for Gz in Pancreatic Islet β-Cell Biology-Reference-Cited by-同舟云学术

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A Role for Gz in Pancreatic Islet β-Cell Biology

Published:2005-09 Issue:36 Volume:280 Page:31708-31713
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Kimple Michelle E.,Nixon Andrew B.,Kelly Patrick,Bailey Candice L.,Young Kenneth H.,Fields Timothy A.,Casey Patrick J.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference49 articles.

1. Pancreatic β-cell growth and survival – a role in obesity-linked type 2 diabetes?

2. Mechanisms of inhibition of insulin release

3. Islet-activating protein. Enhanced insulin secretion and cyclic AMP accumulation in pancreatic islets due to activation of native calcium ionophores.

4. Adrenergic inhibition of insulin secretion involves pertussis toxin-sensitive and -insensitive mechanisms

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1. Gαz-independent and -dependent Improvements With EPA Supplementation on the Early Type 1 Diabetes Phenotype of NOD Mice;Journal of the Endocrine Society;2024-05-21

2. Donor noradrenaline use is associated with better allograft survival in recipients of pancreas transplantation;The Annals of The Royal College of Surgeons of England;2024-01

3. EP3 signaling is decoupled from the regulation of glucose-stimulated insulin secretion in β-cells compensating for obesity and insulin resistance;Islets;2023-07-06

4. Noncanonical Regulation of cAMP‐Dependent Insulin Secretion and Its Implications in Type 2 Diabetes;Comprehensive Physiology;2023-06-26

5. Plasma Prostaglandin E2 Metabolite Levels Predict Type 2 Diabetes Status and One-Year Therapeutic Response Independent of Clinical Markers of Inflammation;Metabolites;2022-12-08

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