N-terminally Truncated WT1 Protein with Oncogenic Properties Overexpressed in Leukemia
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference34 articles.
1. Wilms' tumour: connecting tumorigenesis and organ development in the kidney
2. An internal deletion within an 11p13 zinc finger gene contributes to the development of Wilms' tumor
3. WT1 mutations contribute to abnormal genital system development and hereditary Wilms' tumour
4. Donor splice-site mutations in WT1 are responsible for Frasier syndrome
5. A second transcriptionally active DNA-binding site for the Wilms tumor gene product, WT1.
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1. Evaluating Established Roles, Future Perspectives and Methodological Heterogeneity for Wilms’ Tumor 1 (WT1) Antigen Detection in Adult Renal Cell Carcinoma, Using a Novel N-Terminus Targeted Antibody (Clone WT49);Biomedicines;2022-04-15
2. Sustained AWT1 expression by Dupuytren’s disease myofibroblasts promotes a proinflammatory milieu;Journal of Cell Communication and Signaling;2022-04-12
3. Truncated WT1 Protein Isoform Expression Is Increased in MCF-7 Cells with Long-Term Estrogen Depletion;International Journal of Breast Cancer;2021-11-20
4. WT1 facilitates the self-renewal of leukemia-initiating cells through the upregulation of BCL2L2: WT1-BCL2L2 axis as a new acute myeloid leukemia therapy target;Journal of Translational Medicine;2020-06-24
5. Truncated WT1 protein isoform expression is increased in MCF-7 cells in a long-term estrogen depletion;2019-10-11
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