Sustained AWT1 expression by Dupuytren’s disease myofibroblasts promotes a proinflammatory milieu
Author:
Funder
Canadian Institutes of Health Research
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Molecular Biology,Biochemistry
Link
https://link.springer.com/content/pdf/10.1007/s12079-022-00677-z.pdf
Reference51 articles.
1. Amin EM, Oltean S, Hua J et al (2011) WT1 mutants reveal SRPK1 to be a downstream angiogenesis target by altering VEGF splicing. Cancer Cell 20:768–780. https://doi.org/10.1016/j.ccr.2011.10.016
2. Anuchapreeda S, Rungrojsakul M, Tima S et al (2019) Co-activation of WT1 and AP-1 proteins on WT1 gene promoter to induce WT1 gene expression in K562 cells. Cell Signal 53:339–347. https://doi.org/10.1016/j.cellsig.2018.11.001
3. Anwar MU, Al Ghazal SK, Boome RS (2007) Results of surgical treatment of Dupuytren’s disease in women: a review of 109 consecutive patients. J Hand Surg 32:1423–1428. https://doi.org/10.1016/j.jhsa.2007.06.015
4. Berasain C, Herrero J-I, García-Trevijano ER et al (2003) Expression of Wilms’ tumor suppressor in the liver with cirrhosis: relation to hepatocyte nuclear factor 4 and hepatocellular function. Hepatology (Baltimore, MD) 38:148–157. https://doi.org/10.1053/jhep.2003.50269
5. Bianchi E, Taurone S, Bardella L et al (2015) Involvement of pro-inflammatory cytokines and growth factors in the pathogenesis of Dupuytren’s contracture: a novel target for a possible future therapeutic strategy? Clin Sci 129:711–720. https://doi.org/10.1042/CS20150088
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