Activation of 3-Phosphoinositide-dependent Kinase 1 (PDK1) and Serum- and Glucocorticoid-induced Protein Kinase 1 (SGK1) by Short-chain Sphingolipid C4-ceramide Rescues the Trafficking Defect of ΔF508-Cystic Fibrosis Transmembrane Conductance Regulator (ΔF508-CFTR)-Reference-Cited by-同舟云学术

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Activation of 3-Phosphoinositide-dependent Kinase 1 (PDK1) and Serum- and Glucocorticoid-induced Protein Kinase 1 (SGK1) by Short-chain Sphingolipid C4-ceramide Rescues the Trafficking Defect of ΔF508-Cystic Fibrosis Transmembrane Conductance Regulator (ΔF508-CFTR)

Published:2014-12 Issue:52 Volume:289 Page:35953-35968
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Caohuy Hung,Yang Qingfeng,Eudy Yvonne,Ha Thien-An,Xu Andrew E.,Glover Matthew,Frizzell Raymond A.,Jozwik Catherine,Pollard Harvey B.

Funder

National Institutes of Health

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference58 articles.

1. Cystic fibrosis as a disease of misprocessing of the cystic fibrosis transmembrane conductance regulator protein;Riordan;Am. J. Hum. Genet,1999

2. Chloride channel in the apical membrane of normal and cystic fibrosis airway and intestinal epithelia;Anderson;Am. J. Physiol,1992

3. Digitoxin mimics gene therapy with CFTR and suppresses hypersecretion of IL-8 from cystic fibrosis lung epithelial cells;Srivastava;Proc. Natl. Acad. Sci. U.S.A,2004

4. Defective intracellular transport and processing of CFTR is the molecular basis of most cystic fibrosis;Cheng;Cell,1990

5. A1 adenosine-receptor antagonists activate chloride efflux from cystic fibrosis cells;Eidelman;Proc. Natl. Acad. Sci. U.S.A,1992

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