Inhibition of Ras-GTPase signaling by FPTIII ameliorates development of cardiovascular dysfunction in diabetic–hypertensive rats
Author:
Publisher
Elsevier BV
Subject
Pharmacology,Molecular Medicine,Physiology
Reference32 articles.
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1. Prevention of cardiac dysfunction, kidney fibrosis and lipid metabolic alterations in l-NAME hypertensive rats by sinapic acid—Role of HMG-CoA reductase;European Journal of Pharmacology;2016-04
2. Inhibition of Ras-GTPase Farnesylation and the Ubiquitin-Proteasome System or Treatment with Angiotensin-(1–7) Attenuates Spinal Cord Injury-Induced Cardiac Dysfunction;Journal of Neurotrauma;2011-07
3. Effect of Early Diabetes on the Response to Norepinephrine and Dopamine in Pithed Wistar Kyoto and Spontaneously Hypertensive Rats;Clinical and Experimental Hypertension;2010-10
4. Diabetes mellitus mitigates cardioprotective effects of remifentanil preconditioning in ischemia-reperfused rat heart in association with anti-apoptotic pathways of survival;European Journal of Pharmacology;2010-02
5. FPTIII Mitigates Peroxisome-Mediated Oxidative Stress in Kidneys of Spontaneously Hypertensive Diabetic Rats;Kidney and Blood Pressure Research;2010
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