Signal transduction involving Ras-GTPase contributes to development of hypertension and end-organ damage in spontaneously hypertensive rats-treated with L-NAME-Reference-Cited by-同舟云学术

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Signal transduction involving Ras-GTPase contributes to development of hypertension and end-organ damage in spontaneously hypertensive rats-treated with L-NAME

Published:2005-11 Issue:5 Volume:52 Page:401-412
ISSN:1043-6618
Container-title:Pharmacological Research
language:en
Short-container-title:Pharmacological Research

Author:

BENTER I,FRANCIS I,KHAN I,COJOCEL C,JUGGI J,YOUSIF M,CANATAN H,ALSHAWAF E,AKHTAR S

Publisher

Elsevier BV

Subject

Pharmacology

Cited by 12 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Molecular Signaling Mechanisms of the Renin-Angiotensin System in Heart Failure;Heart Failure: a Companion to Braunwald's Heart Disease;2020

2. Effect of farnesyltransferase inhibition on cardiac remodeling in spontaneously hypertensive rats;International Journal of Cardiology;2013-10

3. Small G Proteins in the Cardiovascular System: Physiological and Pathological Aspects;Physiological Reviews;2013-10

4. Redox Control of GTPases: From Molecular Mechanisms to Functional Significance in Health and Disease;Antioxidants & Redox Signaling;2011-02-15

5. Targeted Genomic Disruption of H-Ras Induces Hypotension Through a NO-cGMP-PKG Pathway–Dependent Mechanism;Hypertension;2010-09

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