Redox Control of GTPases: From Molecular Mechanisms to Functional Significance in Health and Disease-Reference-Cited by-同舟云学术

Redox Control of GTPases: From Molecular Mechanisms to Functional Significance in Health and Disease

Published:2011-02-15 Issue:4 Volume:14 Page:689-724
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Heo Jongyun¹

Affiliation:

1. Department of Chemistry and Biochemistry, The University of Texas at Arlington, Arlington, Texas.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2009.2984

Reference320 articles.

1. Activation of the NADPH oxidase involves the small GTP-binding protein p21rac1

2. S-Glutathiolation of Ras Mediates Redox-sensitive Signaling by Angiotensin II in Vascular Smooth Muscle Cells

3. Oncogenic Ras upregulates NADPH oxidase 1 gene expression through MEK-ERK-dependent phosphorylation of GATA-6

4. Oxidative stress activates extracellular signal-regulated kinases through Src and Ras in cultured cardiac myocytes of neonatal rats.

5. Nitric oxide synthases: structure, function and inhibition

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