Activation of Src tyrosine kinase in microglia in the rat hippocampus following transient forebrain ischemia
Author:
Publisher
Elsevier BV
Subject
General Neuroscience
Reference14 articles.
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2. Identification of microglial signal transduction pathways mediating a neurotoxic response to amyloidogenic fragments of beta-amyloid and prion proteins;Combs;J. Neurosci.,1999
3. Free radicals are involved in continuous activation of nonreceptor tyrosine protein kinase c-Src after ischemia/reperfusion in rat hippocampus;Guo;Neurosci. Lett.,2003
4. Microglial and astroglial reactions to ischemic and kainic acid-induced lesions of the adult rat hippocampus;Jorgensen;Exp. Neurol.,1993
5. Oxidant-induced priming of the macrophage involves activation of p38 mitogen-activated protein kinase through an Src-dependent pathway;Khadaroo;Surgery,2003
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2. c-Src deactivation by the polyphenol 3-O-caffeoylquinic acid abrogates reactive oxygen species-mediated glutamate release from microglia and neuronal excitotoxicity;Free Radical Biology and Medicine;2015-02
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4. Tyrosine phosphorylation of GluK2 up-regulates kainate receptor-mediated responses and downstream signaling after brain ischemia;Proceedings of the National Academy of Sciences;2014-09-08
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